Clinical effects of inhalation of nitrogenīecklake MR, Goldman HI, Bosman AR, Freed CC. Pulmonary effects of acuteĮxposure to nitrous fumes. In: Zenz C, ed.ĭevelopments in occupational medicine. Minutes before anyone enters, and individuals should be equippedĬommins, BT, Raveney FJ, Jesson MW. 4) If possible, enclosed areas should be ventilated for Levels are generally low and within a safe range after 2 weeks,ĭangerous amounts may remain for months if the silo has not been NO((2)) than others, and heavily fertilized crops, cloudyĪnd rain raise the risk of NO((2)) production. Hours after filling, and no one should enter or come in close Suggested safety measures: 1) Silos begin to produce NO((2)) within Industry if farmers are aware of the following dangers and use the ![]() It is possible to prevent this type of exposure in the farm The generation of NO((2)) (3) are also potentially dangerous. Diesel fumes, furnace gases, and chemical processes involving Welding (4), burning cellulose nitrate (5), and dynamite blasting This second pattern appears to respond toįatal and serious exposures to nitrogen oxides are not uniqueįarming but have been reported in association with arc and Biopsies show a bronchiolitis obliterans with Return in three weeks with severe symptoms of fever, chills, and Although these conditions clear spontaneously, illness In another clinical courseĪssociated with silo-filler's disease, exposure causes cough and In fatal exposures, vascular collapse and the outpouring of serum The lungs, where contact with the mucosal moisture produces nitricĪcid, which burns the airways, respiratory bronchioles, and Ifīy smell or sight, the potent nitrogen oxides may be inhaled deep Odorless, and exposure can occur without warning (3). NO((2)) is brown and has an odor, N((2))O((4)) is colorless and They tend to settle in the chute and around the base of the silo,Įxposure often occurs without anyone's entering the silo. Have shown that toxic levels of NO, NO((2)), and N((2))O((4)) are Occupational hazard associated with ensiled crops. Editorial NoteĮditorial Note: The case outlined above is typical for massiveĮxposure to nitrogen oxides. Laboratory Investigations Br, NIOSH, CDC. Mary Imogene Bassett Hospital, Cooperstown, R Rothenberg, MD, StateĮpidemiologist, New York State Dept of Health Immunology Section, Reported by DS Pratt, MD, JJ May, MD, Section of Pulmonary Of symptoms following a recent filling of the silo and histology Illness caused by the inhalation of nitrogen oxides: i.e., rapid Several factors support the diagnosis of silo-filler's disease, Turned on the silo blower and sent another worker up to toss out The farmer reported that, following his farmhand's illness, he had TheĬows were eating normally and producing the usual amounts of milk. Problem with the corn silage, which was still being unloaded. There were no granulomas or hyaline membranes.Īn investigation at the farm 2 weeks later failed to uncover Early bronchiolitis was present no evidence of asthmaĬould be seen. Lungs showed alveoli flooded with proteinaceous material the Weighed 900 gm, and the left weighed 1,000 gm. Lungs with pleural effusions (200 ml) on both sides the right lung Post-mortem examination the next day showed grossly edematous Five hours afterĪdmission, he experienced cardiopulmonary arrest and died despite Not wear an oxygen mask, and remained in shock. Tentative diagnosis of pneumonia was made. The patient was moved to the intensive care unit, where a Tachycardia, and a plain chest radiograph disclosed extensive Showed pH 7.35 PaCO((2)), 32 mm Hg PaO 45 mm Hg and a calculatedīicarbonate of 17.6 mEq/L. White bloodĬount was 31,000 hematocrit, 57.8% and hemoglobin, 18.6 gm.Īrterial blood gas examination while on 2 liters of nasal oxygen ![]() Wheezes and crackles on auscultation of his chest but no signs ofĬonsolidation after asthma was diagnosed, he was treated withĮpinephrine, intravenous aminophylline, and steroids. Respiration, 32 and temperature, 37.5 C (99.5 F). Respiratory distress blood pressure of 84/60 pulse, 128 He made a second attemptĪgain had to climb down and was noted to be cyanotic, pale, andĪt a local hospital, the examining physician noted cyanosis and When the farmhand climbed the chute, he became shortīreath and confused and had to descend. On September 18, he asked aįarmhand to climb up the unloading chute inside the silo and tossįresh silage. In the preceding 10 days, the farm owner had filled this silo Recently filled concrete stave silo and later died of presumed On September 18, 1981, at a farm in Mohawk, New York, aģ9-year-old farmhand was overcome while climbing up the chute of a For assistance, please send e-mail to: Type 508 Accommodation and the title of the report in the subject line of e-mail. Persons using assistive technology might not be able to fully access information in this file.
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